Hopefully we're back on topic. Here's my point. Long-term VLCD can potentially cause adaptations as well. Specifically, I'm talking about T3 and T4 levels. This point is usually oversimplified and used as a reason to avoid VLCDs completely. It's much the same as attempting to equate benign ketosis as being the same as diabetic ketoacidosis. I'm sure you've run into this point in your reading, as I did. I rejected it initially as being non-applicable, or at least of small significance.
Now comes the personal anecdote, take it for what you will.
Two years ago, I developed the symptoms of low T. I had been eating VLC (fat-based) for several months. Since I was aware of the possibility, I developed a hypothesis to increase T, while at the same time preventing lipogenic (fat storage) re-adaptation. It is a result of this that I began the process of carb manipulation, during both bulk and cut cycles, as a way to avoid this specific, hormonal effect. I was successful, all the negative symptoms disappeared, and I was able to resume both my fat loss, as well as my muscle gain. All without lipogenic re-adaptation. Now you understand the points of my earlier posts....
One thing I've not been able to validate, either on a personal basis or by talking to others, is the concept of significant fat gain while in lipolysis. This is usually justified by ASP, but I'm not been able to actually have it happen. When in a significant caloric excess, my fat loss stops, rather than experiencing any regain. If anyone has a different experience, please post, I have a few questions....
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