pre-workout supplements for Cardio,HIIT, etc (not weight training)

[DeeDuub]

Hi there everyone!

I know all the hype about sp250 ,bsn no explode etc bla bla.. everyone knows what im talking about.. the preworkout supplements that give u insane energy ,focus and rage inside the gym.... (ive never used them,just read alota reviews,forums etc )..
anyways

any energy boosters for cardio? I read on a thread somewere that someone used GNC thermoboost for intense running?

Any ideas?
I always run out of energy in running as I want to run long distances ,both for fat loss and hobby but always run outa energy..
(lol i dont suppose NO explode etc woud work rite?)


Please any suggestions?

Thanks

[josh26]

Hi there everyone!


any energy boosters for cardio? I read on a thread somewere that someone used GNC thermoboost for intense running?

Thanks

It's going to be down to the individual, but I noticed the effects of Jack3d far more when doing a warm up and cooldown (rower and treadmill) than when doing lifts.

Those five minutes jogs turned into 20 minutes jogs after the same amount of lifting..

[AMillane7]

Beta alanine and citrulline malate are GREAT before cardio.

Those, along with some BCAAs and a caffeine pill, and you'll be doing great!

[Tobi_DXB]

Beta alanine and citrulline malate are GREAT before cardio.

Those, along with some BCAAs and a caffeine pill, and you'll be doing great!

this^^

for the BCAA I like to use Xtend pre and during cardio, tastes great and also has some citrulline in it. Purple Wraath is good too, just does not taste so good (to me)

[NO HYPE]

any energy boosters for cardio? I read on a thread somewere that someone used GNC thermoboost for intense running? Any ideas? I always run out of energy in running as I want to run long distances ,both for fat loss and hobby but always run outa energy.

Consider the nitrate-mediated oxygen-sparing properties of C-BOL =


Acta Physiol (Oxf). 2007 Sep;191(1):59-66. Epub 2007 Jul 17. Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.
Effects of dietary nitrate on oxygen cost during exercise.
Larsen FJ, Weitzberg E, Lundberg JO, Ekblom B.

AIM: Nitric oxide (NO), synthesized from l-arginine by NO synthases, plays a role in adaptation to physical exercise by modulating blood flow, muscular contraction and glucose uptake and in the control of cellular respiration. Recent studies show that NO can be formed in vivo also from the reduction of inorganic nitrate (NO(3) (-)) and nitrite (NO(2) (-)). The diet constitutes a major source of nitrate, and vegetables are particularly rich in this anion. The aim of this study was to investigate if dietary nitrate had any effect on metabolic and circulatory parameters during exercise. METHOD: In a randomized double-blind placebo-controlled crossover study, we tested the effect of dietary nitrate on physiological and metabolic parameters during exercise. Nine healthy young well-trained men performed submaximal and maximal work tests on a cycle ergometer after two separate 3-day periods of dietary supplementation with sodium nitrate (0.1 mmol kg(-1) day-1) or an equal amount of sodium chloride (placebo). RESULTS: The oxygen cost at submaximal exercise was reduced after nitrate supplementation compared with placebo. On an average Vo(2) decreased from 2.98 +/- 0.57 during CON to 2.82 +/- 0.58 L min(-1) during NIT (P < 0.02) over the four lowest submaximal work rates. Gross efficiency increased from 19.7 +/- 1.6 during CON to 21.1 +/- 1.3% during NIT (P < 0.01) over the four lowest work rates. There was no difference in heart rate, lactate [Hla], ventilation (VE), VE/Vo(2) or respiratory exchange ratio between nitrate and placebo during any of the submaximal work rates. CONCLUSION: We conclude that dietary nitrate supplementation, in an amount achievable through a diet rich in vegetables, results in a lower oxygen demand during submaximal work. This highly surprising effect occurred without an accompanying increase in lactate concentration, indicating that the energy production had become more efficient. The mechanism of action needs to be clarified but a likely first step is the in vivo reduction of dietary nitrate into bioactive nitrogen oxides including nitrite and NO.



J Appl Physiol 107: 1144-1155, 2009. First published August 6, 2009; doi:10.1152/japplphysiol.00722.2009
Stephen J. Bailey,1 Paul Winyard,2 Anni Vanhatalo,1 Jamie R. Blackwell,1 Fred J. DiMenna,1 Daryl P. Wilkerson,1 Joanna Tarr,2 Nigel Benjamin,2 and Andrew M. Jones1
Dietary nitrate supplementation reduces the O2 cost of low-intensity exercise and enhances tolerance to high-intensity exercise in humans

Pharmacological sodium nitrate supplementation has been reported to reduce the O2 cost of submaximal exercise in humans. In this study, we hypothesized that dietary supplementation with inorganic nitrate in the form of beetroot juice (BR) would reduce the O2 cost of submaximal exercise and enhance the tolerance to high-intensity exercise. In a double-blind, placebo (PL)-controlled, crossover study, eight men (aged 19-38 yr) consumed 500 ml/day of either BR (containing 11.2 - 0.6 mM of nitrate) or blackcurrant cordial (as a PL, with negligible nitrate content) for 6 consecutive days and completed a series of "step" moderate-intensity and severe-intensity exercise tests on the last 3 days. On days 4-6, plasma nitrite concentration was significantly greater following dietary nitrate supplementation compared with PL (BR: 273 - 44 vs. PL: 140 - 50 nM; P < 0.05), and systolic blood pressure was significantly reduced (BR: 124 - 2 vs. PL: 132 - 5 mmHg; P < 0.01). During moderate exercise, nitrate supplementation reduced muscle fractional O2 extraction (as estimated using near-infrared spectroscopy). The gain of the increase in pulmonary O2 uptake following the onset of moderate exercise was reduced by 19% in the BR condition (BR: 8.6 - 0.7 vs. PL: 10.8 - 1.6 ml-min-1-W-1; P < 0.05). During severe exercise, the O2 uptake slow component was reduced (BR: 0.57 - 0.20 vs. PL: 0.74 - 0.24 l/min; P < 0.05), and the time-to-exhaustion was extended (BR: 675 - 203 vs. PL: 583 ? 145 s; P < 0.05). The reduced O2 cost of exercise following increased dietary nitrate intake has important implications for our understanding of the factors that regulate mitochondrial respiration and muscle contractile energetics in humans.

The vasodilating properties of dietary nitrates are irrefutable. The clinical use of nitrates indicate that they [significantly] enhance systemic vasodilation. Both nitrate and nitrite can be recycled to produce nitric oxide in a variety of ways. They provide a NOS-independent source of NO, and release NO in an extremely wide distribution of tissues. Nitrite has been investigated as a vasodilator for over 125 years and is a by-product of organic nitrate metabolism.

Based on studies by Tannenbaum 1994; Kelm and Yoshida 1996, nitrates remain in circulation and significantly enhance NO production for around 10 to 16-hours post-administration. Enhanced systemic NO distribution increases blood flow to the muscle itself, thereby enhancing muscle force production/growth/repair.

Dietary nitrate has been shown to reduce diastolic blood pressure in healthy volunteers [Larsen, Ekblom et al. 2006]. Kleinbongard/Dejam et al. 2006, demonstrated that plasma nitrite levels progressively decrease with increasing cardiovascular risk load [risk factors included age, hypertension, high blood cholesterol, and smoking]. It has been proposed that a continuous intake of dietary nitrate may help to maintain tissue levels of nitrite and NO at adequate levels [even when endogneous enzymatic synthesis of NO is disturbed].

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