I was at my local Vitamin Shoppe and noticed that they had X-Factor on clearance for $32 each for 100. I was always curious about it but never pulled the plug on getting it a few months ago and it interests me now.
Can anyone who's ever taken it tell me if it's worth it? And would you recommend it?
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Thread: X-Factor for $32, worth it?
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03-25-2009, 08:22 AM #1
X-Factor for $32, worth it?
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03-25-2009, 08:23 AM #2
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03-25-2009, 08:26 AM #3
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03-25-2009, 08:31 AM #4
- Join Date: Apr 2005
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Just FYI its probably close to or about to expire. They (VS) do this all the time and I remember a few threads in the past about expired X-Factor (from VS) in which Bill said it may loose a tad of potency but its still effective and ok to use.
Last edited by dtrain13; 03-25-2009 at 09:49 AM.
[ANS Performance Representative]
Disclaimer: The above post is my PERSONAL OPINION and DOES NOT REPRESENT the official position of any company or entity. It DOES NOT constitute medical advice.
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03-25-2009, 09:10 AM #5
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03-25-2009, 09:17 AM #6
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03-25-2009, 09:37 AM #7
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03-25-2009, 09:40 AM #8
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03-25-2009, 09:42 AM #9
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03-25-2009, 09:48 AM #10
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03-27-2009, 05:48 PM #11
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03-27-2009, 07:43 PM #12
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03-27-2009, 07:52 PM #13
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$32 is dirt cheap.. definitely worth a shot for that price
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*Disclaimer: The thoughts and opinions of this rep are of his own and does not reflect MET-Rx/Pure Protein as a company. This user is a Bodybuilding.com board representative and is not an employee of MET-Rx/Pure Protein.*
Iron Mulisha Athlete
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03-27-2009, 08:17 PM #14
Yo tmag.. I see you live in bloomfield NJ, is the VS you go to in clifton? I work there.. and just lettin you know they arent out because they are expired but because VS went through what wasnt selling too great and pulled it off the shelves. (now on clearance)
So the exp. date is probably fine, but you can still check it out."I walk slowly, but never walk backward." - Abe Lincoln
"Excuses are the nails that build the House of Failure."
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03-27-2009, 08:22 PM #15
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03-28-2009, 01:01 PM #16
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03-28-2009, 01:03 PM #17
- Join Date: Jan 2006
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buy 2 of them. then use $10 dollar off $50 coupon...
win.[Official] San Diego Brah
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03-28-2009, 01:25 PM #18
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03-28-2009, 04:06 PM #19
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03-28-2009, 04:31 PM #20
Didn't that supp cause cancer in some people?
I personally wouldn't touch it.6'3"
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03-29-2009, 09:01 AM #21
X Factor has never and will never CAUSE cancer in anyone.
I promise you that.
Here is a post I made to prove this point:
CANCER YOU SAY?
Q. Will X Factor Cause Cancer
A. NO
No the ingredient in x factor is not a carcinogen but like other non cancer causing growth promoting agents could be used by an existing tumor
In one study involving the prostate arachidonic acid was observed to cause growth through the PI3K mechanism (PI3K inhibitors blocked growth promoting effects) (18)
PI3K is also the mechanism by which IGF-1 works. So one of the ways x-factor acts as an anabolic is similar to IGF-1.
Other studies have shown stimulation of protein synthesis by arachidonic acid through stimulation of the beta 2 receptor (19, 20) . But stimulation of beta 2 receptors doesn't cause cancer in non cancerous tissues.
Testosterone effects on cancer are analogous to the PI3K and beta 2 mechanisms I just mentioned. It doesn't cause cancer either, but cancer cells will make use of it as a growth promoting agent.
STUDIES ... YAY!
J Nutr. 2004 Dec;134(12 Suppl):3421S-3426S.
Dietary (n-6) PUFA and intestinal tumorigenesis. Whelan J, McEntee MF.
Cancer is the second leading cause of death in the United States, and mortality due to colorectal cancer is only surpassed by lung cancer. Epidemiological studies demonstrate that dietary polyunsaturated fats can have a profound effect on colorectal cancer risk. Experimental data indicate that modulation of cellular (n-6) PUFA metabolism can affect the progression of the disease. This paper discusses the role (n-6) PUFA play in promoting intestinal tumorigenesis and how dietary PUFA from different families interact to modify the neoplastic process. Dietary PUFA that attenuate arachidonic acid metabolism [such as (n-3) PUFA] have antineoplastic properties, whereas those that augment arachidonic acid metabolism, such as linoleic, gamma-linolenic, and arachidonic acids do not appear to enhance tumorigenesis when added to the Western diet but may diminish the beneficial effects of other dietary lipids. It is the relative contributions of the different dietary PUFA that may determine overall risk for and progression of the disease.Cancer. 1999 Sep 15;86(6):1019-27.
Association of energy and fat intake with prostate carcinoma risk: results from The Netherlands Cohort Study.
Schuurman AG, van den Brandt PA, Dorant E, Brants HA, Goldbohm RA.
Department of Epidemiology, Maastricht University, Maastricht, The Netherlands.
BACKGROUND: The roles of energy and fat intake as risk factors for prostate carcinoma are still questionable. Therefore, these factors were evaluated in the Netherlands Cohort Study described in this article. METHODS: The cohort study consisted of 58,279 men ages 55-69 years at baseline in 1986. After 6.3 years of follow-up, 642 incident prostate carcinoma cases were available for analysis. Intake of energy, fat, and separate fatty acids were measured by means of a self-administered questionnaire; fat intake was adjusted for energy by regression analysis. The case-cohort method was used to calculate rate ratios (RRs). Analyses were conducted for all prostate carcinoma cases together as well as for case subgroups (latent vs. nonlatent and localized vs. advanced). RESULTS: No associations were found in multivariate analyses between prostate carcinoma and intake of energy, total fat, total saturated fatty acids, or total trans unsaturated fatty acids (RR highest vs. lowest quintile: 0.99, 1.10, 1.19, and 0.99, respectively). Oleic acid intake showed a nonsignificant positive association (RR = 1.38, 95% CI: 0.88-2.19). Positive associations were also observed for intake of oleic acid in subgroup analyses. Linoleic (RR = 0.78, 95% CI: 0. 56-1.09) and linolenic (RR = 0.76, 95% CI: 0.66-1.04) acid intake were associated with nonsignificantly decreased risks; only for linolenic acid did these associations persist in subgroup analyses. No associations were found for intake of arachidonic acid, eicosapentaenoic acid, or docosahexaenoic acid. CONCLUSIONS: These data suggest that certain fatty acids might be involved in prostate carcinoma occurrence, although the possibility that these were chance findings cannot be ruled out. Copyright 1999 American Cancer Society.Bull Cancer. 2005 Jul;92(7):670-84. Related Articles, Links
[Dietary fatty acids and colorectal and prostate cancers: epidemiological studies]
Astorg P. UMR Inserm 557/INRA/CNAM Epidemiologie nutritionnelle, Institut scientifique et technique de l'Alimentation, Conservatoire national des Arts et Metiers, 5 rue du Vertbois, 75003 Paris. pierre.astorg@cnam.fr
OBJECTIVE: This study reviews epidemiological works having studied the associations of dietary fatty acids, especially of n-6 or n-3 polyunsaturated fatty acids (PUFA), with the risks of colorectal and prostate cancers. METHODS: The epidemiological studies reviewed were those having tested the association of colorectal and prostate cancer risk with the dietary intake or the blood or adipose tissue levels of fatty acids, especially of n-6 and n-3 PUFA, and with the dietary intake of fish and seafood. RESULTS: Most studies based on a dietary questionnaire did not find any association of the risk of colorectal cancer with the consumption of either total fatty acids or any particular fatty acid, after adjustment for total energy intake had been made. A few studies suggest that trans fatty acid consumption could increase colorectal cancer risk. Most studies based either on a dietary questionnaire or on biomarkers, did not find any association of total, saturated or monounsaturated fatty acid, as well as of linoleic or arachidonic acids, with prostate cancer risk, after adjustment for total energy intake. Most studies failed to find an association of prostate cancer risk with fish or long-chain n-3 PUFA intake, but recent cohort studies did find an inverse association of fish consumption with the risk of the latest stages of prostate cancer. In contrast, alpha-linolenic acid intake was associated with an increase of prostate cancer risk in a majority of epidemiological studies, but other studies did not find this association. This latter point might be of concern, and needs to be clarified by other results, especially those of ongoing prospective studies.Am J Clin Nutr. 2004 Jul;80(1):204-16. Related Articles, Links
Dietary intake of n-3 and n-6 fatty acids and the risk of prostate cancer.
Leitzmann MF, Stampfer MJ, Michaud DS, Augustsson K, Colditz GC, Willett WC, Giovannucci EL.
Nutritional Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD 20892, USA. leitzmann@mail.nih.gov
BACKGROUND: Laboratory studies have shown that n-3 fatty acids inhibit and n-6 fatty acids stimulate prostate tumor growth, but whether the dietary intake of these fatty acids affects prostate cancer risk in humans remains unclear. OBJECTIVE: We prospectively evaluated the association between intakes of alpha-linolenic (ALA; 18:3n-3), eicosapentaenoic (EPA; 20:5n-3), docosahexaenoic (DHA; 22:6n-3), linoleic (LA; 18:2n-6), and arachidonic (AA; 20:4n-6) acids and prostate cancer risk. DESIGN: A cohort of 47866 US men aged 40-75 y with no cancer history in 1986 was followed for 14 y. RESULTS: During follow-up, 2965 new cases of total prostate cancer were ascertained, 448 of which were advanced prostate cancer. ALA intake was unrelated to the risk of total prostate cancer. In contrast, the multivariate relative risks (RRs) of advanced prostate cancer from comparisons of extreme quintiles of ALA from nonanimal sources and ALA from meat and dairy sources were 2.02 (95% CI: 1.35, 3.03) and 1.53 (0.88, 2.66), respectively. EPA and DHA intakes were related to lower prostate cancer risk. The multivariate RRs of total and advanced prostate cancer from comparisons of extreme quintiles of the combination of EPA and DHA were 0.89 (0.77, 1.04) and 0.74 (0.49, 1.08), respectively. LA and AA intakes were unrelated to the risk of prostate cancer. The multivariate RR of advanced prostate cancer from a comparison of extreme quintiles of the ratio of LA to ALA was 0.62 (0.45, 0.86). CONCLUSIONS: Increased dietary intakes of ALA may increase the risk of advanced prostate cancer. In contrast, EPA and DHA intakes may reduce the risk of total and advanced prostate cancer.
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Joey - 10/16/2011
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03-29-2009, 09:32 AM #22
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