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  1. #1
    Registered User kezvanbeck's Avatar
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    How Many eggs a day is safe?

    How many eggs is it actually safe to eat daily? Whole eggs not just the Whites, because I'm basically seeing eggs as my quickest way for protein but I don't want to over do it-.- and will I get the full amount of protein raw? I just find it quicker raw with milk.. Thanks!
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  2. #2
    hi Hyruliangoat's Avatar
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    about tree fitty.


    raw eggs are NOT as bioavailable as cooked eggs. it takes like 2 minutes to cook.
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    I don't think there's a clear, solid consensus on what that number is, but it's definitely better to eat them cooked.

    I personally eat 5. Crack 'em into a bowl, scramble then up, then microwave.
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    y u lok here 2canman's Avatar
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    around 6 i'd say. I'm probably wrong though, but it's just the amount i'd eat in a day, if i did eat eggs.
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    well my uncles Cardiologist makes him eat atleast 2 eggs a day to for his heart... I went to the dr about 3 weeks ago and had bloodwork done and he said my cholesterol was a little high and I told him I eat 4 eggs a day and he said to cut it down or only eat eggbeaters.

    I was confused because I had always read that the cholesterol in eggs in associated with the cholesterol that is considered bad. He just told me to do cardio, a lot.
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    Registered User kezvanbeck's Avatar
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    Originally Posted by jammyo40 View Post
    I don't think there's a clear, solid consensus on what that number is, but it's definitely better to eat them cooked.

    I personally eat 5. Crack 'em into a bowl, scramble then up, then microwave.
    Alright thanks! Yeah scrambled in the microwave is easy but don't like it that way, personally I like hard boiled, but raw is so convenient haha, but if cooked will get me more outta em then I'll cook as often as I can be bothered lol.. so you think 5-10 a day is safe? 10 being a maximum! I see people that have like over 20 a day, I can't see that being good for you:|
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    I usually eat 3-4 in the morning, depending on how hungry I am. Cooked is definitely better (protein wise) than raw, but you'll also decrease the risk of you getting sick and being out for a week. But, other than that, eat away! You'll have to make sure your hitting your caloric/macro needs in other foods, though.
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    IFBB Amateur, M. Physique ArchangelEST's Avatar
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    I think you'll reach your own tolerance level before you reach the "dangerous" levels off egg consumption.(cooked eggs - don't recommend uncooked at all) Even if you could eat 30+ eggs a day, I'm pretty sure you'd get real sick of them, real fast.
    I suggest any quantity that allows you to keep them in your menu for an extended period of time if you plan on getting a portion of your nutrients from them.

    I used to eat 10 eggs a day. After about a month I got so sick of them, that I had to literally quit for a few months before I could tolerate the taste again. Now I cap myself at about 5 large eggs a day and this way I like them just fine and actually enjoy them.

    Another user is trying 24 eggs a day at another thread. Might follow his progress and see how crappy he feels after a while. I don't think many people have tried an almost all Egg diet before.
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    some people will eat a dozen a day. So it's probably a safe bet that you can eat quite a bit.


    If you're looking for quick protein, look into Greek Yogurt.
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    As many as will fit your macros. In the summer, I ate close to eight-ten whole eggs a day.
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    Your eating them raw?

    Brb salmonella..lulz
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    team ketchup AdamWW's Avatar
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    I would assume you could eat just as many eggs and you could chicken, beef, or fish to the tune of the same macro-nutrient content..

    IE - if you can eat a 30oz t-bone steak, you can probably eat 20 eggs all the same.. no they are not the same macros, but they both have a lot of fat and high calorie counts..

    For myself, if I eat more than 3 per day i get terrible gas..
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    Registered User kezvanbeck's Avatar
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    Originally Posted by ErikTheElectric View Post
    Your eating them raw?

    Brb salmonella..lulz
    The thing issss, salmonella is actually found on the shell.. like the bird s**t, so as long as I dont sit licking the actual eggs, and make sure theyre clean, and dont handle the actual egg when cracked with my hands, after touching the shell, im sure i wont die and on the packaging, it also states the hens that have layed these eggs have all been treated with some medicine to prevent salmonella, so ill be cool i think....
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    Registered User kezvanbeck's Avatar
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    Thanks everyone for your advice! Its always good when people give actual advice, not cocky ignorant comments -.- I think ill just stick to eating around about 5 eggs a day, I do prefer raw but if i can get , more protein out of them cooked then its a no brainer ain't it! and then after time maybe up my intake, theyre just a lot more convenient when theres no quick alternative in!
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    i eat 3 to 5 a day....cooked.
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    I eat 8-10 every day.....I love eggs and it is where I get the majority of my daily fat.
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    Originally Posted by Hyruliangoat View Post
    about tree fitty.


    raw eggs are NOT as bioavailable as cooked eggs. it takes like 2 minutes to cook.
    this. just hardboil like 24 eggs every few days
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    Originally Posted by kezvanbeck View Post
    How many eggs is it actually safe to eat daily? Whole eggs not just the Whites, because I'm basically seeing eggs as my quickest way for protein but I don't want to over do it-.- and will I get the full amount of protein raw? I just find it quicker raw with milk.. Thanks!
    As many as you can fit into your macros. Eggs are perfectly healthy, they're nature's multivitamin. Why would there be a maximum safe limit?

    Are the Current Dietary Guidelines Regarding Egg Consumption Appropriate?

    http://jn.nutrition.org/content/134/1/187.full

    CONCLUSION: If judged as a whole food, and not simply as a source of dietary cholesterol, the positive contribution of eggs to a healthy diet becomes apparent. Because eggs are a conventional food containing nutrients that play fundamental roles beyond basic nutrition, their promotion as a functional food should be considered. This discussion has examined the possible role of egg nutrients in the prevention and treatment of specific symptoms associated with chronic age-related diseases. Furthermore, evidence has been presented showing that the current blanket recommendations regarding dietary cholesterol and egg intake are unwarranted for the majority of people and are not supported by scientific data.

    Fernandez ML. Dietary cholesterol provided by eggs and plasma lipoproteins in healthy populations. Curr Opin Clin Nutr Metab Care. 2006 Jan;9(1):8-12.

    PURPOSE OF REVIEW: Extensive research has not clearly established a link between egg consumption and risk for coronary heart disease. The effects of egg intake on plasma lipids and low-density lipoprotein (LDL) atherogenicity in healthy populations need to be addressed. RECENT FINDINGS: The lack of connection between heart disease and egg intake could partially be explained by the fact that dietary cholesterol increases the concentrations of both circulating LDL and high-density lipoprotein (HDL) cholesterol in those individuals who experience an increase in plasma cholesterol following egg consumption (hyperresponders). It is also important to note that 70% of the population experiences a mild increase or no alterations in plasma cholesterol concentrations when challenged with high amounts of dietary cholesterol (hyporesponders). Egg intake has been shown to promote the formation of large LDL, in addition to shifting individuals from the LDL pattern B to pattern A, which is less atherogenic. Eggs are also good sources of antioxidants known to protect the eye; therefore, increased plasma concentrations of lutein and zeaxanthin in individuals consuming eggs are also of interest, especially in those populations susceptible to developing macular degeneration and eye cataracts. SUMMARY: For these reasons, dietary recommendations aimed at restricting egg consumption should not be generalized to include all individuals. We need to acknowledge that diverse healthy populations experience no risk in developing coronary heart disease by increasing their intake of cholesterol but, in contrast, they may have multiple beneficial effects by the inclusion of eggs in their regular diet.

    Kritchevsky SB. A review of scientific research and recommendations regarding eggs. J Am Coll Nutr. 2004 Dec;23(6 Suppl):596S-600S.

    For much of the past 40 years, the public has been warned away from eggs because of a concern over coronary heart disease risk. This concern is based on three observations: 1. eggs are a rich source of dietary cholesterol; 2. when fed experimentally, dietary cholesterol increases serum cholesterol and; 3. high serum cholesterol predicts the onset of coronary heart disease. However, data from free-living populations show that egg consumption is not associated with higher cholesterol levels. Furthermore, as a whole, the epidemiologic literature does not support the idea that egg consumption is a risk factor for coronary disease. Within the nutritional community there is a growing appreciation that health derives from an overall pattern of diet rather than from the avoidance of particular foods, and there has been a shift in the tone in recent dietary recommendations away from "avoidance" messages to ones that promote healthy eating patterns. The most recent American Heart Association guidelines no longer include a recommendation to limit egg consumption, but recommend the adoption of eating practices associated with good health. Based on the epidemiologic evidence, there is no reason to think that such a healthy eating pattern could not include eggs.

    Herron KL, Lofgren IE, Sharman M, Volek JS, Fernandez ML. Metabolism. 2004 Jun;53(6):823-30. High intake of cholesterol results in less atherogenic low-density lipoprotein particles in men and women independent of response classification.

    The influence of a high-cholesterol diet on the atherogenicity of the low-density lipoprotein (LDL) particle was examined by measuring LDL peak diameter and composition, LDL susceptibility to oxidation, and the distribution of cholesterol between LDL subclasses. The crossover intervention randomly assigned 27 premenopausal women and 25 men (18 to 50 years) to an egg (640 mg/d additional dietary cholesterol) or placebo (0 mg/d additional dietary cholesterol) diet for 30 days, followed by a 3-week washout period. Subjects were classified as either hyperresponders (>2.5 mg/dL increase in plasma cholesterol for each 100 mg additional dietary cholesterol consumed) or hyporesponders to dietary cholesterol. Sex was found to have a significant effect on 3 of the parameters examined. LDL peak diameter was significantly larger (P <.005) in females (26.78 +/- 0.59 nm, n = 27) as compared with males (26.52 +/- 0.49 nm, n = 25), regardless of response to dietary cholesterol. The LDL particles of the male participants also had a higher number of triglyceride (TG) and cholesteryl ester (CE) molecules (P <.01); however, cholesterol ester transfer protein (CETP) activity was higher in females (P <.05). Response classification also revealed significant differences in the determination of LDL subclasses. Independent of sex, the LDL-1 particle (P <.05), which is considered to be less atherogenic, was predominant in hyperresponders and this finding was associated with increased cholesterol intake (interactive effect, P <.001). In addition, CETP and lecithin: cholesterol acyltransferase (LCAT) activities were higher in hyperresponders during the egg period (interactive effect, P <.05). Sex, response to cholesterol intake, and diet were not found to affect the susceptibility of LDL to oxidation (P > 0.5). Because LDL peak diameter was not decreased and the larger LDL-1 subclass was greater in hyperresponders following egg intake, these data indicate that the consumption of a high-cholesterol diet does not negatively influence the atherogenicity of the LDL particle.

    Herron KL, Vega-Lopez S, Conde K, Ramjiganesh T, Shachter NS, Fernandez ML. Men classified as hypo- or hyperresponders to dietary cholesterol feeding exhibit differences in lipoprotein metabolism. J Nutr. 2003 Apr;133(4):1036-42.

    The purpose of this study was to evaluate the differences that occur within the plasma compartment of normolipidemic men, classified on the basis of their response to prolonged consumption of additional dietary cholesterol. Using a crossover design, 40 men aged 18-57 y were randomly allocated to an egg (640 mg/d additional dietary cholesterol) or placebo group (0 mg/d additional dietary cholesterol), for two 30-d periods, which were separated by a 3-wk washout period. Subjects were classified as hypo- [increase in plasma total cholesterol (TC) of <0.05 mmol/L for each additional 100 mg of dietary cholesterol consumed] or hyperresponders (increase in TC of > or =0.06 mmol/L for each additional 100 mg of dietary cholesterol consumed) on the basis of their plasma reaction to the additional dietary cholesterol provided. Male hyporesponders did not experience an increase in LDL cholesterol (LDL-C) or HDL cholesterol (HDL-C) during the egg period, whereas both lipoproteins were significantly (P < 0.0001 and P < 0.05, respectively) elevated in hyperresponders. Although the LDL/HDL ratio was increased in male hyperresponders after the high cholesterol period, the mean increase experienced by this population was still within National Cholesterol Education Program guidelines. Furthermore, male hyperresponders had higher lecithin cholesterol acyltransferase (P < 0.05) and cholesteryl ester transfer protein (P < 0.05) activities during the egg period, which suggests an increase in reverse cholesterol transport. These data suggest that additional dietary cholesterol does not increase the risk of developing an atherogenic lipoprotein profile in healthy men, regardless of their response classification.

    Eggs and heart disease risk: perpetuating the misperception

    http://www.ajcn.org/content/75/2/333.full

    "The fact that no studies in the past decade have reported a significant relation between either egg consumption or dietary cholesterol intakes and heart disease risk (5) is consistent with the view that the hypothesis that dietary cholesterol is a risk factor for heart disease should be dismissed."

    Conclusion: Whole eggs = gooooood.
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    I eat as many as my pet hen will lay, we are seeing who quits first.
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  20. #20
    Southern Brah theculture's Avatar
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    I eat 5 every morning (2 yolk and 3 white).......
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    Registered User nuketurnal's Avatar
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    i only eat 2 whole, 3 whites.. and i gas a lot
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    I'm cooking at a new breakfast place, and I eat about 2 cups of egg whites and around 10 whole eggs a day there. It's great how much free protein I can get there. Sometimes I even eat pork sirloin. Let's not forget it's a pancake breakfast place so I get amazing carbs.
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    Registered User TopStarUSA's Avatar
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    At one point in my life I was eating 9 whole eggs a day...I am still alive
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    Originally Posted by Hyruliangoat View Post
    raw eggs are NOT as bioavailable as cooked eggs. it takes like 2 minutes to cook.
    How significant is the difference in bioavailability?
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    Originally Posted by 2canman View Post
    I'm probably wrong though,.
    this
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    I eat 8.
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    I eat 10-12 whole eggs a day. 6 in the morning scrambled and then 4-6 at night with dinner.
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    Our best bet: wait for that guy who eats 24 eggs a day to follow the diet for a prolonged period - say 6 weeks, then get a blood test - then we will know how many eggs a day is safe.

    Being dead serious, no ethics committee would allow studies to force feed participant eggs just to determine the maximum amount of eggs that can be safely consumed a day, this is the best chance we've got .
    Last edited by FourToEight; 09-20-2011 at 03:04 AM.
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    Eggs are good.

    Eggs with no other fats in the diet is not best.

    Also bloodwork and checking with your doc about familial hypercholesterolemia is a plus.
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    Around 25 seem to be safe:

    Normal Plasma Cholesterol in an 88-Year-Old Man who Eats 25 Eggs a Day: Mechanisms of Adaptation (1991) Kern Jr, New England J Medicine 324(13):896-899

    The effect of dietary cholesterol intake on serum cholesterol level is known to vary among individuals. The homeostatic and regulatory mechanisms which tend to keep serum cholesterol constant operate at different levels of efficiency in different individuals. An extreme case is reported here. A physician and colleagues from the University of Colorado School of Medicine have studied an 88-year-old man who, for ill-defined psychological reasons, has consumed 20 to 30 eggs daily for at least 15 years. This individual has maintained normal serum lipid levels and has no history of clinically important heart disease. He consented to participate in a variety of tests of cholesterol metabolism; the findings were compared with those obtained in an ongoing study of 11 normal volunteers who were tested while consuming normal diets and diets supplemented with five eggs per day. It was found that this individual had extremely efficient mechanisms that compensated for his phenomenally high cholesterol intake. In particular, he absorbed only 18 % of the cholesterol that he consumed; the comparison subjects absorbed 54.6% when on low-cholesterol diets and 46.4% when on high-cholesterol diets. He also showed a doubling of the usual rate of conversion of cholesterol to bile acids, moderately reduced cholesterol synthesis, and a possible increase in biliary cholesterol secretion. "These physiologic adaptations would leave little if any of the dietary cholesterol to elevate plasma cholesterol levels and be deposited in arterial walls."
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