that link apparently no longer works, probably because of the amount of spam post the very long thread attracted. Unfortunate because it was a very good thread.
here is the actual post.
dosing reccomendations repost/compilation (still needs a little updating and work) for puffy nipples, gynecomastia and pectoral feminization
small update: as a point of clarification I highly reccomend use of AI/prolactin suppressor in most cases since there is generally benefit regardless of "need". In cases where there is ANY associated fat topical yohimbine( ie Yohimburn ES) ALONG with needed caloric restriction is reccomended. In all cases even where fat is not an issue calorie moderation, if not restriction, is something that I reccomend.
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ONE, not all, of these aromatase inhibitor protocols (added for clarification).
in the case of letrozole protocol, tapering or following with either aromasin or AIFM . Low dose AIFM can be used directly on the nipples in conjuction with either aromasin or letrozole (so thats kind of an exception to the one AI rule)
aromasin/exemestane protocol (min 6-8 weeks)
25mg ED taken with a high fat meal (lower doses may be taken if you experience dry joints or lethargy which indicate too much estrogen suppression, though its somewhat uncommon with aromasin)
AIFM protocol (min 6-8 weeks)
2-4 pumps per day
direct application to nipples and surrounding tissue is beneficial
see
www.afboard.com for more specifics and to ask questions (I have used it but you will get more and fuller answers there)
(as with aromasin, if you experience dry joints or lethargy dosing should be cut back)
Letrozole/femara protocol
2.5mg ED 2-3 weeks followed by either aromasin or AIFM. You can also insert such a 2-3 week "hyper suppression" period into the middle of either aromasin or AIFM. Some people may use longer. If gyno is aggravated, use of letrozole initially is more often indicated. Dry joints, lethargy and libido loss are common, which is why this protocol is generally kept short.
Cabaser/cabergoline Protocols (min 6-8 weeks)
front load protocol
(I prefer this but its not a necessity), I highly reccomended if you have "active gyno"- aggravated and painful or if you are lactating/have fluid discharge.
1mg 3-5 days, .5mg for 5-7 days, .5mg every other day after 2 months reccomend .5mg every third day (unless you have severe and blood work indicated hyperprolactinemia)
slow and low protocol
.5mg eod
Yohimburn ES (topical yohimbine)
Use as needed, I generally reccomend use only prior to exercise. Though you will find other opinions. Its only needed if there are local fat deposits.
OTHER DOPAMINERGICS that may be used:
Bromocriptine, Selegiline and Pramipexole.
Levodopa is not reccomended.
Selegiline may be used in conjuction with bromocriptine, cabergoline or pramipexole, though dosing may need to be adjusted
important factors in success:
1. calorie deficit
2. avoiding or at least reducing alcohol, marijuana, opiates (preferably avoiding)
3. Avoiding large insulin spikes (eliminate high fructose corn syrup and hydrogenated oils, watch sugar, fructose and saturated fats)- dont eat massive simple carb meals.
4. Avoiding environmental estrogens- dont drink from heated plastic, avoid using products with LAVENDER, Tea trea oil products MAY be an issue (there is some dispute, but at this time I reccomend against using any product with tea tree and a "body" products has it). There are more, I will have to find the list and post again.
5. Dont use designer steroids while treating gynecomastia, they have unknown binding and there impacts are very difficult to control in this area.
6. steroids like masteron and proviron may be used. Unless taking testosterone its probably not wise to use winstrol.
7. avoid overheating, high heat is strongly linked to prolactin release
8. avoid dehydration, for the above reason and because dehydration can cause other hormone spikes.
this is kind of a partial list, but it contains most of the common and important factors.
If you are taking SSRI's then cabergoline may be of additional benefit, since it has been shown to help with the libido issues they cause. SSRI's have also been linked to prolactin issues, so they can be a factor (dont stop taking them, just offset their impact.
repost. I would also like to reiterate the importance of tips 1-4. these can be make or break aspects when treating. You can suppress all the right hormones but if you are producing insulin and igf like gangbusters you will see little catabolism or apoptosis of tissue (certainly not of fat). and eliminating your own estrogen, but having it replaced by environmental or supplemental exposure can be a big hold up. Now there are some phytoestrogens that MAY help things along, but on the whole you want to avoid exogenous estrogens as much as possible. I am still on the fence as to whether lignans and their enterolactone/enterodiol metabolites are a plus or minus. and things that you may not realize can contain potent estrogens, ginsing for instance depending on the area cultivated and extraction method. see below.
Exp Biol Med (Maywood). 2004 Jun;229(6):560-8. Links
Mycotoxins in root extracts of American and Asian ginseng bind estrogen receptors alpha and beta.Gray SL, Lackey BR, Tate PL, Riley MB, Camper ND.
Department of Plant and Environmental Sciences, Clemson University, South Carolina 29634, USA.
sngry@clemson.edu
The estrogenic activity of ginseng has been the subject of conflicting reports. Cell proliferation, induction of estrogen-responsive genes, and isolated cases of adverse reactions such as postmenopausal vaginal bleeding and gynecomastia have been reported after ginseng treatment. Other studies report antiproliferative effects with no induction of estrogen-responsive genes. We developed estrogen receptor (ER) alpha and ER alpha competitive binding assays using recombinant receptors and [(3)H]-17 alpha-estradiol to detect phytoestrogens in extracts of Asian ginseng root (Panax ginseng C. A. Meyer) and American ginseng root (Panax quinquefolius L.). Root extracts contained substances that bound both receptor isoforms. These substances had a two to three times greater affinity for ER alpha. Significantly higher binding was found in methanol extracts than in hot water extracts. Subsequent analysis of the extracts revealed significant ER binding attributable to zearalenone, the estrogenic mycotoxin produced by several Fusarium species. The ER showed no binding affinity for Rb1 and Rg1, the major ginsenosides found in P. quinquefolius and P. ginseng, respectively. Thus, ginseng extraction methods, plant species tested, and mycotoxin contaminants may help to explain the disparate literature reports. The prevalence and health significance of fungal contamination in herbal products used for medicinal purposes should be further investigated.
Gyno Help Please..
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